Gastrointestinal (GI): NSAIDs that block COX-one activity have correlations with GI complications due to inhibition of your synthesis of gastroprotective brokers which include PGE2 and other prostaglandins. This inhibition results in reduced mucin production by gastric epithelial cells, significantly less bicarbonate secretion, and fewer epithelial cell turnover, among the other https://arthurr431fcg5.webbuzzfeed.com/profile